Using a humanized rodent model, the researchers transplanted a small RNA proton well known as short hairpin RNA (shRNA), that prompted RNA division in to human red blood branch cells to stop the countenance of CCR5 in human defence cells.
The commentary yield justification that this plan can be an in effect approach to provide HIV-infected individuals, by call long-term and fast rebate of CCR5.
Saki Shimizu, Patrick Hong, Balamurugan Arumugam, Lauren Pokomo, Joshua Boyer, Naoya Koizumi, Panyamol Kittipongdaja, Angela Chen, Greg Bristol, Zoran Galic, Jerome A. Zack, Otto Yang, Irvin S.Y. Chen, Benhur Lee and Dong Sung An, all of UCLA.
Blood, Journal of the American Society of Hematology
The investigate was saved by a Rheumatology Fellowship Training grant, the UCLA AIDS Institute, the UCLA Center for AIDS Research, the National Institute of Allergy and Infectious Diseases, the National Heart, Lung and Blood Institute, and the National Cancer Institute.
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